The Herpes simplex virus (HSV) is a member of the Herpesviridae family, of which two types are known: Type 1 (HSV-1) and Type 2 (HSV-2) which presentslight antigenic differences. HSV-1 causes chiefly oral-facial lesions, while HSV-2 is mainly responsible for genital lesions, but this distinction is not binding, asboth types may occasionally cause infection in either anatomical site. HSV may also cause a form of ocular cheratitis, and lesions of the central nervous system.
The primary infection is often in a subclinical form and is rarely diagnosed. After a latency period of variable duration, reactivation may occur and viral replication may give rise to clinical lesions. HSV infection is an important cause of infant morbidity and mortality, and thus it is important to determine the immune status of women during pregnancy in order to detect serum conversion. The assay of IgM is also important for the diagnosis of neonatal infection and encephalitis caused by HSV infection contracted during birth.
| Product | Cat # | Description | Specimen material | Method | Size |
| Herpes simplex virus 1 IgG | E-HSG-K05 | Qualitative detection of Anti – HSV 1 IgG antibodies | Serum and Plasma | ELISA | 96 tests |
| Herpes simplex virus 2 IgG | E-HHG-K06 | Qualitative detection of Anti- HSV 2 IgG antibodies | Serum and Plasma | ELISA | 96 tests |
| Herpes simplex virus 1&2 IgG | E-HGG-K24 | Qualitative detection of Anti- HSV 1& 2 IgG antibodies | Serum and Plasma | ELISA | 96 tests |
| Herpes simplex virus 1&2 IgM | E-HGM-K25 | Qualitative detection of Anti- HSV 1& 2 IgM antibodies | Serum and Plasma | ELISA | 96 tests |
Sample Volume : 10 µl
Controls/ Calibrators : 3 Controls
Incubation : 45’+ 45’+15 min
Substrate : TMB
